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Carlton M. Bates MD

  • Associate Professor, CHP/Pediatrics/Nephrology
Research Interests
  • Bladder
  • Urothelium
  • Apoptosis
  • Cyclosphosphamide
  • Bladder injury
  • Fibroblast growth factor receptor
  • FGF7
  • Mouse
  • Conditional knockout
Representative Publications
  1. Narla ST, Bushnell DS, Schaefer CM, Nouraie M, Tometich JT, Hand TW,  Bates CM. Loss of fibroblast growth factor receptor 2 (FGFR2) leads to defective bladder urothelial regeneration after Cyclophosphamide injury. Am J Pathol, 2020, in press. PMCID-pending
  2. Hurley E, Peoples ES, Bates CM, Hunstad DA, Barkin SL. Perspective commentary from the Society for Pediatric Research: supporting early-stage pediatric physician-scientist success. Pediatr Res, 87(5):834-838, 2020. PMID: 31896124, PMCID: pending.
  3. Anslow MJ, Bodnar AJ, Cerqueira DM, Bushnell D, Shrom BE, Sims-Lucas S, Bates CM and Ho J. Increased rates of vesicoureteral reflux in mice from deletion of Dicer in the peri-Wolffian duct stroma. Pediatr Res, 88:382-390. 2020. PMID: 32015493, PMCID: PMC7396288.
  4. Narla ST, Bushnell DS, Schaefer CM, Nouraie M, Bates CM. Keratinocyte Growth Factor reduces injury and leads to early recovery from Cyclophosphamide bladder injury. Am J Pathol, 190(1):108-124, 2020. PMID: 31654363, PMCID: PMC6943803.